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Tion, and return home healthy.John D Grabenstein deputy director Military Vaccine Agency, 5113 Leesburg Pike, Suite 402, Falls Church, VA 22041 USA john.grabenstein@us.army.mil William Winkenwerder Jr assistant secretary of defense (health affairs) Washington, DC, USA5 Eckart RE, Love SS, Atwood JE, Arness MK, Cassimatis DC, Campbell CL, et al. Incidence and follow-up of inflammatory cardiac comp
E data: CGT. Wrote the manuscript: RHuang, XL, RHolm, JMN, CGT, ZS. All authors read and approved the final manuscript. All authors read and approved the final manuscript. Acknowledgments This study was financially supported by grants from Inger and John Fredriksen Foundation and Radium Hospital Research Foundation and The Norwegian Cancer Society. We thank Ellen Hellesylt, Mette Synn e F sund, M
Ovary [38], indicating a possibly different role of ALDH1 in ovarian cancer. Our present immunohistochemical study of 248 wellcharacterized patients showed high levels of ALDH1 expression in ovarian carcinoma cells, which were observed in 15.7 of the total cases, was associated with early-stage tumor, well-differentiated tumor and better survivals, although ALDH1 was not an independent risk fac
The rise of CSC theory, some specific CSC markers were being discovered to identify putative CSCs, and ALDH1 is becoming to act as a CSC marker in the CSC studies of variable tumor types in vitro and in vivo. Epithelial-to-mesenchymal transition (EMT) is an important driver of tumor invasion and metastasis, which may be a feature of CSC. Compared to ALDH1(-) EMT cells, only ALDH1(+) EMT cells had
Cancer Res. 2014;74(13):3579?0. 12. Mizuno T, Suzuki N, Makino H, Furui T, Morii E, Aoki H, et al. Cancer stemlike cells of ovarian clear cell carcinoma are enriched in the ALDH-high population associated with an accelerated scavenging system in reactive oxygen species. Gynecol Oncol. 2015;137(2):299-305. 13. Yasuda K, Torigoe T, Morita R, Kuroda T, Takahashi A, Matsuzaki J, et al. Ovarian cancer
Icine, Faculty of Medicine, University of Oslo, Oslo, Norway. 3Departments of Gynaecology, The Norwegian Radium Hospital, Oslo University Hospital, Oslo, Norway. 4Departments of Gynaecology, Institute of Clinical Medicine, Faculty of Medicine, University of Oslo, Oslo, Norway. Received: 27 January 2015 Accepted: 25 JuneReferences 1. Eskander RN, Tewari KS. Incorporation of anti-angiogenesis thera
Fferentiation. Cell Cycle. 2009;8(20):3297?02. 9. Chute JP, Muramoto GG, Whitesides J, Colvin M, Safi R, Chao NJ, et al. Inhibition of aldehyde dehydrogenase and retinoid signaling induces the expansion of human hematopoietic stem cells. Proc Natl Acad Sci U S A. 2006;103(31):11707?2. 10. Ginestier C, Hur MH, Charafe-Jauffret E, Monville F, Dutcher J, Brown M, et al. ALDH1 is a marker of normal a
Cancer Res. 2014;74(13):3579?0. 12. Mizuno T, Suzuki N, Makino H, Furui T, Morii E, Aoki H, et al. Cancer stemlike cells of ovarian clear cell carcinoma are enriched in the ALDH-high population associated with an accelerated scavenging system in reactive oxygen species. Gynecol Oncol. 2015;137(2):299-305. 13. Yasuda K, Torigoe T, Morita R, Kuroda T, Takahashi A, Matsuzaki J, et al. Ovarian cancer
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